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Thread: AIDS Treatment development!

  1. #1 AIDS Treatment development! 
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    i heard that there is no cure for AIDS but will there at least be a vaccine or any other method that will slow down the effect of AIDs taking place? Answers needed! Thanks in advance!


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  3. #2  
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    I just read a small article about that recently, I guess they've developed a vaccine which attacks AIDS in a new way. Which of course is great, because if you can attack it using different mechanisms your chance of beating it goes up considerably. I can check more into it when I get home from work if no one else has posted much by then.


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    Forum Freshman Tranquil's Avatar
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    I think, making a vaccine would take a long way . . . We all know, there have been and always are attempts to develop a vaccine, but because of high mutation rate of the virus, it is unseccessfull. And there is also one more think to be aware of - HIV has many cells for reproduction (macrophages, T-lymphocytes and others cells). Because of large variety of receptors to attach (CCR4, CXCR5, coreceptor CD4 etc.) It would be hard to find a method of developing vaccine . . .
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    A vaccine is probabily on the way, but you must be beware, that it will not be 100 % proof
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    I read in another thread that this disease can be slowed down considerably already.
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    Quote Originally Posted by casperl82
    I read in another thread that this disease can be slowed down considerably already.
    Yes, the tritherapy have changed the evolution of HIV infection
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    How exactly? Do you mean the rate of spread of the virus or the rate of its progression in an individual? And do you just mean that this is a consequence of taking drugs, as in the virus would return to normal if tritherapy was stopped now?
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    Quote Originally Posted by Robbie
    How exactly? Do you mean the rate of spread of the virus or the rate of its progression in an individual? And do you just mean that this is a consequence of taking drugs, as in the virus would return to normal if tritherapy was stopped now?
    It does not change the rate of spread

    It's almost stop the progression of the infection in an individual. The people under tritherapy have a normal amount of the white LT4 cell, and an amount of virus very law

    a scientific study, have tried to do a therepeutic window (stop the treatement for some month) and it was a failure. Whenever you try to stop the treatment the infection will evolve again.
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    Fair enough, another thing, you know when patients may have leukaemia and have chemo/radiotherapy in order to kill off their WBCs and may then be administered a bone marrow transplant, could a similar treatment be used for AIDS since you would kill off the infected cells then reintroduce new, "clean" cells.

    I realise the danger & the fact this would not be 100% effective but then again, its a similar scenario for leukaemia patients.
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    Quote Originally Posted by Robbie
    Fair enough, another thing, you know when patients may have leukaemia and have chemo/radiotherapy in order to kill off their WBCs and may then be administered a bone marrow transplant, could a similar treatment be used for AIDS since you would kill off the infected cells then reintroduce new, "clean" cells.

    I realise the danger & the fact this would not be 100% effective but then again, its a similar scenario for leukaemia patients.
    I think that the HIV virus do not only exist in leukocyt. Even if you destroy all the LT4 of the patient, the HIV will still be present.
    That's said that was a nice idear
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  12. #11  
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    A virus isn’t a living particle, it uses our own cell machinery to reproduce itselfs. Hence we can not kill it only prevent it from reproducing.

    see

    http://www.tibotec.com/bgdisplay.jht...=HIV_discovery

    This is Belgian firm which does research on HIV medicines.

    preview in quote:
    Four main events in the HIV life cycle offer leads for the development of antiviral therapies. Viral Entry is an interplay between viral factors and host cells. Upon entry into the cell, the virus is uncoated and its RNA genome is converted into DNA by HIV reverse transcriptase. The resulting proviral DNA is integrated into the host cell genome by HIV integrase. Once integrated, host factors can transcribe the proviral DNA into new viral RNA genomes, and translate this RNA into precursor polyproteins. These polyproteins are cleaved into smaller pieces by HIV protease, providing the building blocks for new viruses. After assembly and maturation, new viruses are released from the infected cell to initiate subsequent rounds of infection.
    he who forgets...will be destined to remember (Nothing Man - Pearl Jam)
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